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Furthermore, because we indicated previously that nSP induced different cellular responses from submicron-sized silica particles, such as those described above, it was of interest to analyse the effects of particle size on osteoclast differentiation.
As we indicated previously, no sequence similarity can be detected between MAEL and DEAD helicase domains.
We understand that 24 h time point may not identify preceding or proceeding events as we indicated previously [ 7- 9].
As we indicated previously, different chemicals could alter the levels of survivin-2B, IKK alpha and P73 along with the changes in autophagy.
We understand that using 4 weeks-instillations only may not identify preceding or proceeding events as we indicated previously [ 38- 40].
Based on our study, where samples were collected as long as 7 days after exposure, it is indicated that the BNF is continuously supplied from other organs (e.g. liver) to the intestinal cells, similarly to what we indicated previously for the BNF effect observed on intestinal CYP1A.
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In addition, all of these studies consider a static scenario with a fixed number of users in the system, whereas in our study, we consider, as indicated previously, a dynamic setting where, again, (data) users come to the system and leave it after a finite duration, corresponding to the transfer of their files.
As indicated previously, we selected historical releases of the GeneLynx database [10] as the source for annotation over an extended time period.
We have, also, implemented, as indicated previously, an admission control scheme based on a maximal number smax of active users in the system; smax is obtained by setting a minimal threshold nmin on the number of subcarriers per admitted user.
We did not have confidence in the 30% figure as indicated previously.
In both cases we dropped founder genotype probabilities that did not sum to at least 4.8 as indicated previously.
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