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In step 2, we identified duplicate records.
We identified duplicate gene pairs with sufficient divergences that represent this antagonism relationship in the yeast S. cerevisiae.
We identified duplicate publications for exclusion by examining the study name, authors, study population, location, and the dates of duration of the study.
We identified duplicate genes and singletons in a genome based on an all-against-all BlastP alignment (Altschul et al. 1997).
We identified duplicate subjects with the AmpFLSTR Profiler Plus® PCR amplification kit (PE Applied Biosystems, Foster City, CA) to type nine tetranucleotide short tandem repeat loci and the Amelogenin locus.
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We identified duplicates and overlaps which revealed that, on average, each of the eight groups gathered 16.9 requirements resulting in 2.4 distinct requirements per active participant.
In this way, we identified duplicated regions that might have been misassembled or erroneously merged in the current genome assembly.
Below, we describe how we identified duplicated genes, HT genes, and synonymous and nonsynonymous SNPs and then describe statistical analyses applied to each type of variant.
In all tables and figures, we identified duplicated loci by /i or /ii, regardless of whether both loci were added to the map.
To distinguish among these processes and to examine the generality of these patterns, we identified duplicated genes in nine sequenced Drosophila genomes.
We identified duplicated and relocated genes in multiple sequenced Drosophila genomes to examine the evolutionary dynamics of gene traffic between X chromosomes and autosomes.
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