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The observed IQR for arm sample size in our empirical data is 51 to 270, and to represent moderate and large studies we generated studies with n ~ U 50, 150) and n ~ U 150, 300).
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We generated study strains (see Table 1) that carried a qC1 -balanced mel-28 allele and were also homozygous for the putative genetic interactor.
To achieve a precise normalization, we generated a study-specific SPECT template using both SPECT and MR scans of all patients and normal controls under study following a procedure described in detail elsewhere [ 21].
To visually assess the mean change estimates and corresponding 95% confidence intervals across studies, we generated forest plots and grouped studies by dose of alcohol.
We generated multiple simulation studies using binary predictors.
In our simulations, we generated case-control studies each including a single disease SNP.
By using a two stage model of carcinogenesis, we generated Monte Carlo studies to assess the efficiency and robustness of the 3-poly test for animal carcinogenicity experiments.
In addition, there are discrepancies of phenotypes between Cthrc1 transgenic mice which we generated in this study and the transgenic mice overexpressing Cthrc1 previously reported [14].
All the clones we generated in this study produced both type 1 and type 2 cytokines: IFN-γ, TNF-α, IL-13 and in some cases IL-10.
The data we generated in this study are available at http://sites.google.com/site/jjlmmtf.com/site/jjlmmtf
The results we generated in this study are all based on PET (0.4 µm pore size) membranes.
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CEO of Professional Science Editing for Scientists @ prosciediting.com