However, in order to explain the high levels of Cd, Pb and Ni detected in samples bought at the local market, we could hypothesize that herbs were harvested in contaminated sites, since their collection area was unknown.
In the latter case, we could hypothesize that a unified modeling approach should be able to reproduce, and to explain, the structural and the dynamic features of collaborations in different domains.
For instance, we could hypothesize that the removal of sugars from glycosphingolipids in the membrane alters its structure in such a way that it constitutes an early step in carbohydrate partitioning.
Taking inspiration from other medical fields, we could hypothesize a combined regimen in which biologic agents are administered simultaneously at a low or ultra-low dosage, targeting several pathogenetic mechanisms but avoiding important side effects.
We could hypothesize a future clinical use of the networks' perspective in extracting PCS and DAN metabolic values from standard ROIs to identify patients at risk, using only one FDG-PET scan, and possibly to treat before clinical manifestation of cognitive fog or cancer fatigue occurs.
Therefore, if we consider our results within this model, we could hypothesize that the highest incidence of muscle injuries displayed by football players with AA genotype could be due to acidic intracellular environment created by muscle activity due to the elevated lactate transport from arterial blood into the muscle fibers for its oxidation, as noted in the Cupeiro at al. study [10].
Alternatively, we could hypothesize that the normal faults of the Crocker fault system are the manifestation of extension at the crest of an anticlinorium that is forming above a deep, convex-upward thrust fault that farther updip is producing the offshore fold-and-thrust belt (Fig. 5b).
Taken together the data showed up to now, we could hypothesize that fibroblast activation can also take place in vivo.
We could hypothesize IL-10 implication for maintaining STAT3 phosphorylation at a high level in Daudi and Namalwa resistant cells.
We could hypothesize that these differences arise from the adaptation of molecular parameters, such as codon usage, polypeptide folding and resistance to inactivation by mammalian cell proteases.
Thus, we could hypothesize that the secretion-defective mutant proteins are either incorrectly folded or have altered electrostatic surfaces, which could affects LGI1 export.
