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Now we begin to determine the optimal sector number and accomplish airspace partition via using a GA based on integer code.
However, only by performing detailed investigations involving selective cell isolation, clonal analysis together with cell labelling and tracking studies, will we begin to determine the true existence of a mesothelial stem cell.
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In addition to examining the independent associations of AAP and SHS with the methylation and expression of our candidate genes, we looked to see if we could begin to determine the relative impacts of the two exposures on mechanisms of the immune system.
We also begin to determine the subtle reasons why published waveforms from older AC and DC monitors appear to differ so greatly.
Yet, we can begin to determine when the preponderance is relatively more developmental compared to environments that are more obstructionist or more predatory.
By continuing to assess these characteristics in vivo we can begin to determine the genetic correlates of host range and specialization and, therefore, shed light on understanding factors that influence variation in arbovirus evolution and activity in nature.
Thus, by understanding the mechanisms of this infection, we can begin to determine how to target this component of viral CNS infection therapeutically to eradicate this significant viral reservoir.
We can now begin to determine the extent to which apical and basal VNO subsystems regulate functionally distinct behavioral repertoires.
We could then begin to determine the specific AMF strategies favourable in an agricultural context and ask what selection pressures facilitated their spread in the AMF community.
We next used a histological approach to begin to determine the underlying mechanism for the defect in ET function that we observed.
As scientists begin to determine how the nocebo effect works, we would do well to use their findings to manage that most 21st-century of all diseases – anxiety.
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