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In light of the wide range of self-selected glycemic index foods, we are unable to explain the rise in HDL-C in the CHO-LCD group.
For example, we are unable to explain the association between heart disease and primary headache.
Here due to space constraints we are unable to explain the dataset.
We are unable to explain how the anisotropic NPs were generated, and this topic will be explored in future work.
Readers may ask for more details but due to space constraints we are unable to explain the dataset.
Although we are unable to explain our contradictory findings, many factors may have contributed to increased postoperative pain, in addition to sympathetic tone activation [12].
We are unable to explain these results, yet we cannot rule out that the model was unstable, which would introduce a bias.
However, we are unable to explain why smaller holes such as 100- or 50-nm diameter were not filled with PDMS.
The contrary intuition is that the occurrence of $M$ is a paradigm case of something that we are unable to explain.
We are unable to explain why there was no perennial with an rufipogon-type organelle genomes because currently there is no evidence that the organelle genomes of O. meridionalis confers any advantage over the Asian O. rufipogon type.
To date, we are unable to explain this variation between nuclei of the same muscle.
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