In the two data sets of the G6PD region we analyzed, Sabeti et al. (2002)'s and Verrelli et al. (2002)'s data, no new mutations at closely linked loci were detected.
Using the same isotope data we analyzed, Darimont et al. [14] also found evidence supporting the niche variation hypothesis (estimated using isotopic variability, island wolves exhibited both the largest niche width and greatest among-individual variation; Fig. 1).
That compares to 43% of the genes, we analyzed from Hsieh et al. (2011) and 73% of the genes we analyzed from Wolff et al. (2011).
We analyzed whether Eschrich et al. [ 2] 43 genes survival predictor recognized specifically any of our tumor subtypes.
For further confirmation, we analyzed the Cam et al. dataset [ 16] of profiles of growing and serum starved T98 breast cancer cells.
To investigate whether luminal-simple-classified luminal A tumors also showed lower FGA in independent data sets, we analyzed the Chin et al. [ 3] aCGH data set by using the genomic-subtype classification from corresponding gene-expression data in the combined Affymetrix data set.
To evaluate whether miR-34c and the other 4 members of the miRNA family have an essential role in the first cleavage division both in vivo and in vitro, we analyzed miR-34b/c (Choi et al., 2011) and miR-449 (Bao et al., 2012) knockout mice, and also generated miR-34b/c; miR-449 double knockout (herein called miR-dKO) mice.
For the north Adriatic Sea, we analyzed data from Lotze et al. [113], who used a multidisciplinary approach to assess the ecological changes and overall shift in diversity over historical time scales in 12 estuaries and coastal seas worldwide, including the north Adriatic Sea.
For ookinetes that did fully differentiate, we analyzed their motility (Moon et al., 2009) and found that gliding motility in Δ shlp1 ookinetes was indistinguishable from that of WT parasites, indicating that their motor function was unimpaired.
We analyzed data from Hartman et al.'s study of IFN-induced STAT binding and mRNA transcription for chromosome 22 [ 16], and to our surprise, we observed a marked probe level correlation between STAT1 binding and differential expression in response to IFN-γ.
