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T-IhNSCs' growth rate was stable over passaging and so was their strict growth factor dependence.
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Moreover, the deletion was stable over passage of d1BTV in mammalian cells.
Third, although these pathways show distinct profiles in PDX models compared with original primary tumors, they appear to be stable over passages.
The proportions of the different cell types was stable over time (from passage 18 to 25), indicating that relevant percentages of cells within the serially passaged hNSC bulk population grown as neurospheres display a molecular and immunochemical signature closely resembling that described for OPC populations isolated from the human CNS by means of immunopanning or FACS sorting [4], [5], [45].
This morphology was stable over several cell passages.
The telomere length of each mutant expressing the fusion protein was stable over the successive passages.
The growth of these cells was stable over long-term culture as seen by the maintenance of cell numbers upon serial passage and was similar to growth seen in normal fibroblasts (Figure S2).
Samples of LuCaP 35 and LuCaP 49 at five different passages were analyzed by DNA arrays to examine if gene expression was stable over time.
Nonetheless, the association was stable over time.
Mean birth weight was stable over time.
The RA, OA, and NL groups clustered separately at all passages, confirming that the RA methylome signature is stable over many passages (see Figure 1A for representative examples).
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