The activity was inhibited to 0.017, that is, the inhibition rate was 93.4%%.
In the presence of hBD3, RANTES was inhibited to a similar extent to the inhibition afforded by the endosomal inhibitor chloroquine (CQ) (Fig. 4D).
At 400 MOI infection of Ad-MIP, cell growth of HCT116-p53+/+ cells was inhibited to approximately 50% of control, whereas no inhibition of HCT116-p53−/− cells was detected (Fig. 6).
Under salinity stress, plumule growth was inhibited to a greater extent than radicle growth.
Only FIII-3 was inhibited to some extent by TPCK, which is a specific inhibitor for chymotrypsin-like serine proteases.
Conversely, resorption by osteoclasts derived from long bones was inhibited to a greater degree by the cysteine proteinase inhibitor.
The lower i-C4/n-C4 ratiof of the hydrous experiments plus U-ore indicated that the carbocation mechanism was inhibited to some extent. .
Conversely, copper deposition occurred in other areas under conditions where nucleation proceeded, but grain growth was inhibited to produce a fine, homogeneous microstructure.
The rate of proliferation was inhibited to 82 and 12% in parental and CD133 + sphere forming cells, respectively on treatment with 10 μM concentration of mefloquine.
The resorptive activity of scapular osteoclasts was inhibited to a greater extent by the MMP inhibitor CT-1746 than by the cysteine proteinase inhibitor E64.
By comparison, papain was inhibited to 50% at a molar ratio of 2 CS:1 papain and 1.5 CT:1 papain.
