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An identical, nonsense mutation, W129X, was characterised in two unrelated patients.
Delirium was characterised in two different ways: (1) as a binary variable (never vs ever delirious), and (2) as a categorical variable (0 3 days of delirium).
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This paper investigates the evolution of the gene tra, which was characterised in twelve tephritid species belonging to the less extensively analysed genus Anastrepha.
Baseline morbidity was characterised in three ways: (1) history for each of cancer, large vessel disease, renal failure and visual deterioration; (2) the number of primary care contacts in the year prior to the index date; and (3) Charlson comorbidity index.
Three missense mutations were characterised in three cases of NHL after screening exons 5-8 of p53 of all the tumours with single-strand conformation polymorphism (SSCP) analysis.
Abnormal sensory experience is characterised in three ways: (1) hypersensory sensation triggered by stimuli that is sub-threshold for triggering that sensation, (2) pesthesia altered sensory experience, and (3) allodynia sensation triggered by stimuli that do not normally trigger those sensations.
Furthermore the expression profile of these genes was characterised in-vitro in both monolayer and three dimensional (3D) cultures of tendon fibroblasts to identify whether such models fully recapitulate the tendon phenotype.
The culture response to oxidative stress, produced either by addition of exogenous hydrogen peroxide (H2O2) or by high dissolved oxygen tension (DOT), was characterised in terms of the activities of two key defensive enzymes: catalase (CAT) and superoxide dismutase (SOD).
The culture response to oxidative stress, produced either by menadione (MD) or by H2O2, was characterised in terms of the intracellular activities of two key defensive enzymes, catalase (CAT) and superoxide dismutase (SOD), in the presence or absence of a range of vitamin supplements.
Ten years later, a new Tnt1-like element, Retrolyc1, was characterised in Lycopersicon peruvianum [ 13].
To date, p16 INK4a germline mutations were characterised in only two out of 55 MM patients less than 30 years, but that both had a history of familial melanoma (Whiteman et al, 1997; Tsao et al, 2000).
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