After hydrocortisone the left hippocampal decrease in BOLD signal was attenuated with the magnitude of attenuation correlating with the increase seen after hydrocortisone infusion.
More strikingly, Aβ42 aggregation in the hybrids-treated Drosophila was attenuated with effective neuroprotection.
The pressure drop was attenuated with increasing temperature for a given superficial gas, and base and pulse superficial liquid velocities.
Changes in HR tended to be similar across running speeds; however, the magnitude of effects was attenuated with increases in BWS.
The head motion was restricted with foam padding around the head and the noise of the scanner was attenuated with earplugs.
The beam was attenuated with an acoustic-optical filter to the energy level of 6.25nJ per pulse at the focal plane of the microscope objective.
However, this effect was attenuated with major fluid resuscitation and here renal blood flow reached levels comparable to the conscious state [54].
The detrimental effect of curvature was attenuated with a large initial kernel and by increasing the thickness of the mixing layer.
Treatment with the stable thromboxane A2 mimetic U46619 increased ROK-dependent Thr855 phosphorylation of MYPT that was attenuated with the ROK antagonist Y-27632; representative data from n = 3.
In the present study, we saw very large to extremely large reductions in VO2 from 0 to 10% BWS and from 10 to 20% BWS; however, the effect, while still moderate to large in magnitude, was attenuated with additional BWS between 20 and 30% BWS.
Most dramatically, the capacity of Au[(Gly-Trp-Met)2B] and Au[(Met)2B] to elicit ROS generation disappeared while the ability of Au[(Gly-Tyr-TrCys)2B], Au[(Gly-Tyr-Met)2B] and Au[(TrCys)2B] to elicit an oxidative stress response was attenuated, with a significant difference in responses, as measured statistically.
