Sentence examples for vulnerability to cell from inspiring English sources

The French government in May banned the use of cell phones in elementary schools citing the 'principle of precaution' regarding children's vulnerability to cell phone radiation.

These results suggest that EA, by inhibition of the Wnt/β-catenin pathway, compromised an important survival signal in CLL cells and increased their vulnerability to cell killing induced by chemotherapeutic agents.

However, it is of interest to note that Sub mice showed an increased number (although not significant) of very large sized adipocytes (i.e. larger than 91 µm in diameter) in the perigonadal pad, which can be considered as an incipient hypertrophic obesity [48], [49] possibly leading to increased vulnerability to cell death [50].

The brain is largely postmitotic, and proteins may well be co-opted to serve other functions which, when lost, disrupt neurophysiology and increase vulnerability to cell death.

The role of MAPK p38 is divergent, because MAPK p38 pathways may be involved in anti-apoptotic processes (Roberts et al., 2000), but may also increase the vulnerability to cell death (Aroor and Shukla, 2004).

Despite the similarity in cell type between the CA1 and CA3 regions, evidence suggests that these regions show different vulnerabilities to cell death depending on the stressor 2. For example aging-dependent complications such as cardiovascular disease 3, stroke 4, and decreased cerebral blood flow 5 can increase neuronal ischemic damage in the hippocampus.

Aging is associated with increased vulnerability to hippocampal cell death 1.

The neonatal stage of life is characterized by a high sensitivity to pain and a great vulnerability to neuronal cell death [ 1].

In PC12 cells, ALDH2 deficiency produced by introducing ALDH2 ∗ 2 gene leads to marked accumulation of 4-HNE in response to oxidative stress stimuli and increased vulnerability to 4-HNE-induced 4-HNE-induced 4-HNE-induced

This intriguing finding raises the possibility that oblation of the energy-sensing LKB1-AMPK axis, while conferring biosynthetic and proliferative advantages, also imparts vulnerability to the cells so that they are hypersensitive to energetic crisis-induced killing.

At present, we cannot explain why GalR1 antagonist administration results in increased vulnerability to kainate-induced cell death in C57BL/6 mice.