P-gp has also been proposed as a regulator for Cl− selective channels with a role in cell volume regulation [52].
It demonstrates that rapid modifications of actin cytoskeleton dynamics regulated by specific signaling pathways play a functional role in cell volume regulation.
Volume-regulated anion channels (VRAC) are widely expressed chloride channels that are critical for the cell volume regulation.
Such an increase likely results from renal impairment associated with volume regulation abnormalities, suggesting a potential role for ADM in volume regulation after liver transplantation.
These cells express different complements of transport proteins involved in volume regulation.
Background: Glycine betaine is important in cell volume regulation and in remethylating homocysteine, a vascular risk factor.
We obtained 3D Brillouin maps of cells in 2D and 3D microenvironments, which reveal mechanical changes due to cytoskeletal modulation and cell volume regulation.
The biophysical and pharmacological properties of this Cl- channel are consistent with a role in triggering volume regulation in lymphocytes exposed to hyposmotic conditions.
The goal of this study was to conduct a subject-specific evaluation of a control-theoretic plasma volume regulation model in humans.
Since the early days of cell volume regulation research, the role of actin cytoskeleton organization and rearrangement has attracted specific interest.
Little information is available on the complex pathophysiology of volume regulation after blood donation.
