Sentence examples for vitro treated with from inspiring English sources

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(B and C) iWAT of Adrb1−/−; Adrb2−/−; Adrb3−/−mice or control mice (Adrb1+/−; Adrb2+/−; Adrb3+/+) was in vitro treated with 100 μmol/L norepinephrine (NE) or control PBS.

For tumour establishment, GBM cells were in vitro treated with Wnt3a (30 ng/ml for 5 days) and then injected subcutaneously (5 × 10 cells) in a 200- μl total volume into both dorsolateral flanks.

Human SZ95 sebocytes in vitro treated with hormone levels that can be found in 60-year-old women produce less lipids than sebocytes treated with a hormone mixture representing that found in serum of 20-year-old women.

Representative dot blots showing synthesis of IFN-γ by CD4+ and CD8+ T cells isolated from diestral bitches and in vitro treated with DMSO (control) or 30, 300 and 3000 ng/ml of aglepristone, are presented on Figure  1.

To evaluate whether changes induced by 5-AZA-CdR on the immune profile of cancer cells could make them more sensitive to the host's immune recognition, TS/A cells were in vitro treated with 1  μℳ 5-AZA-CdR and tested by phenotypic and functional assays.

Based on these results, to evaluate whether modulation of the immune profile induced by 5-AZA-CdR was effective in increasing immune recognition of cancer cells, cytolytic activity of P1A-specific CTL was measured against TS/A untreated or in vitro treated with 1  μℳ 5-AZA-CdR, pulsed or not with P1A peptide.

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We cultured primary mouse BVECs in vitro, treated them with GDF11 or vehicle, and measured pSMAD2/3 levels.

A-549 cells were cultured in vitro and treated with different dosages of SCF and cell morphology was observed.

Based on the effect of different-sized ZnO nanoparticles in the absence/presence of UV irradiation on SMMC 7721 cell lines, it was evident that all these ZnO nanoparticles have the similar inhibition capacity on target cancer cells, and UV irradiation could greatly enhance this inhibition effect on SMMC-7721 cells in vitro when treated with ZnO nanoparticles.

Murine macrophages infected with L. braziliensis in vitro were treated with DETC as described above.

Additionally, we confirmed that knocking-down ERRα lead to similar genomic effects demonstrated in vitro when treated with the ERRα specific antagonist.

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