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A number of in vitro studies indicates a role for PTEN signaling in neuronal size, dendritic and axonal branching and neuronal polarization [8], [9].
Moreover, evidence from in vitro studies indicates that metformin also has effects on cell proliferation via intracellular targets other than AMPK [ 100, 101].
Growing evidence, largely derived from in vitro studies, indicates that platelet activation reflected by increased levels of PF4 and release of polyphosphates adversely affects fibrin clot properties including formation of compact networks [ 14, 37].
Substantive evidence from animal (in vivo and in vitro) and human in vitro studies indicates that VA and its metabolites (particularly retinoic acid (RA)) have a powerful role in the regulation of both innate and adaptive immune responses [ 13, 14].
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In vitro studies indicated that silver nanoparticles caused apoptosis in mouse embryonic stem cells [38].
Furthermore, in vitro studies indicated that methanobactin can act as an oxygen radical scavenger [19, 20].
All in vitro studies indicated that codeine's permeability is higher than metoprolol's.
In vitro studies indicated that most of these compounds are relatively potent inhibitors of each enzyme.
Evaluation of humanised 1A3B-7 (Hu1A3B-7) in in vitro studies indicated that Hu1A3B-7 retained both broad specificity and neutralising activity.
In vitro studies indicate that enhancing cell proliferation with growth factor exposure enhances plasmid DNA uptake and expression.
Previous in vitro studies indicated that Atazanavir (ATV) has a distinct resistance profile than other protease inhibitors (PIs).
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CEO of Professional Science Editing for Scientists @ prosciediting.com