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Results in vitro show that expression of CXCR4 is regulated by PAX3-FKHR in Rhabdomyosarcoma cell lines [ 60] and by Pax7 in C2C12 cell lines [ 13].
Furthermore, antitumor assay in vitro show that β-CD-HPG/DTX effectively inhibited proliferation of human breast adenocarcinoma cells.
Studies in vitro show that DENV induces DC activation and maturation [19], [20]; however, the profile of activation/maturation differs between in vitro-infected and non-infected bystander DC [14], [20], [21], [22].
Video microscopy analyses of axons in vitro show that subtypes of vesicular organelles display different kinetic behaviors ranging from very rapid, relatively continuous anterograde and retrograde movements that span long distances to infrequent short range bidirectional movements separated by short or very long pauses [41], [42].
Studies in vitro show that oestrogen rapidly up-regulates telomerase gene expression and activity [ 38].
Studies performed in vitro show that these inhibitors either on their own or accompanied by an antibiotic, facilitate dissolution of biofilms or prevent their formation [ 66– 66].
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Cell culture in vitro shows that expression of Hdac6 and deacetylated tubulin are associated with tumorigenesis, cellular motility and cancer cell migration and invasion (Rey et al., 2011).
The results in vitro showed that DS-L.
The release result in vitro showed that BC ST P123 MMs presented sustained release behavior compared to control group.
Results in vitro showed that putrescine modification enabled a dramatic increase in binding of Aβ to the plaques.
The AT1 receptor-binding assays in vitro showed that all the synthesized compounds had nanomolar affinity for the AT1 receptor.
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