Sentence examples for vitro models data from inspiring English sources

We found no enhancement of growth inhibition following treatment with a combination of ICR62 with afatinib or gemcitabine or erlotinib in these in vitro models (data not shown).

Additional microarray experiments performed in parallel with NM reference strain revealed similar gene expression patterns in vivo compared to in vitro models (data not shown) indicating that the trends described by the Dutch 602 outbreak field strain are likely to prove relevant to other C. burnetii strains more generally.

As a further control, we have tested the migration of A375 (a commercially available melanoma cell line) in our model and we found similar transmigration ability if compared to what was previously reported by other authors for the same cell line in a similar in vitro model (data not shown) [ 53].

Therefore, there is an urgent need to validate existing in vitro models using data from animal models, although these animal models do not fully simulate the physiology of humans.

Both of these compounds were effective at inhibiting HUVEC growth and the formation of microtubules in in vitro models of angiogenesis (unpublished data).

Although precise reasons for leveraging use of in vitro models for generation of genomics data are perhaps elusive, the practical aspects cited above for use of in vitro systems undoubtedly play a role.

This study provides an in vitro model and baseline data on future developments of new strategies for salivary gland regeneration and replacement.

Still, due to the limitations of the chosen in vitro model system our data need to be interpreted carefully.

Data from in vitro models have not always matched those from observational or clinical studies.

Although the step from in vitro models to man is large, these data do provide a framework for understanding how a high FAAH-IR, by reducing the local endocanninoid levels, is associated with disease outcome in cases with a mid-range expression of CB1IR – in the cases with a low CB1IR, the target for endocannabinoid action is absent.

Importantly, data from in vitro models and from retrospective series in women with breast cancer suggest that both EGFR and HER2 overexpression confer resistance to hormonal therapy [ 2].