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Evidence for a number of viruses in vitro indicates that lower antibody concentrations are required to inhibit infection propagated by free virus than are required to inhibit infection propagated by cell-to-cell spread.
Most recently, evidence in vitro indicates that there is a recapitulation of embryonic hedgehog signalling in acute epithelial injury and chronic inflammation, a finding with key implications for inflammatory disorders of the intestine, such as inflammatory bowel diseases.
As seen in our experiments, previous work in neonatal rats in vitro indicates that activation of mACh receptors on XII MNs causes depolarization, facilitates repetitive firing and potentiates XII inspiratory burst output [33], [34].
Suppression of enhanced neurite growth by the antibody to BDNF in vitro indicates that BDNF was upregulated in injured neurons and glia in the DRG and can be released into culture medium to promote neurite growth via autocrine mechanisms [50].
Our work in vitro indicates that CaMKII is specifically phosphorylated by Wnt5a signaling (our unpublished observations).
Available evidence in human populations and human cells in vitro indicates that the prostate is a target for inorganic arsenic carcinogenesis.
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Hemolytic activity in vitro indicated that EPSAH was non-hemolytic.
The results of transfection study in vitro indicated that LPCM exhibited higher gene expression than PIC.
Results of preliminary antifungal tests against Trichophyton rubrum in vitro indicated that most of the synthesized compounds showed excellent activities.
Circular dichroism and a degradation assay in vitro indicated that PCFSs had good stability and a low degradation rate.
The results of the colony forming units (CFU) plate counting in vitro indicate that the adhesion of Staphylococcus epidermidis (SE) to PET is suppressed by silver coating.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com