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The results presented here reveal that cultivation of E. faecalis in blood in vitro has a profound impact on its transcriptome, which includes a number of virulence traits.
Activity-dependent cell loss in vitro has a characteristically slower time course than observed in vivo.
We first demonstrated that curcumin, in vitro, has a role in the regulation of proliferation and apoptosis of MDA.MB231 cells.
These data indicate that increasing ROS in vitro has a major effect not only in limiting T-cell survival but also in reducing T-cell function.
We have demonstrated that, despite the generally low level of microglial CD45RO, triggering this molecule with an antibody (UCHL-1) in vitro has a profound effect in human microglia.
For example, preclinical studies have shown that — compared with paclitaxel — docetaxel is a stronger promoter of tubulin polymerisation in vitro, has a longer intracellular half-life and demonstrates greater activity in some tumour models (Barasoain et al, 1991; Ringel and Horwitz, 1991; Bissery et al, 1995).
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Thus, identifying senescent cells in in vivo and in vitro has an important diagnostic and therapeutic potential.
The well-rooted plantlets obtained in vitro had a 94% survival rate after acclimation.
Somatic cells in vitro have a finite life expectancy before entering a state of senescence, but it is unclear whether this state occurs in vivo in kidney development, growth, and aging.
Recent work by Lombardi et al. [31] showed that the waiting times between neuronal avalanches recorded in vitro have a distribution with an initial power-law regime.
Human coronary endothelial cells treated with nebivolol in vitro have a decrease in VCAM-1, ICAM-1, E- and P-Selectin expression [48].
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