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To corroborate the in vitro data, we also examined the effect of PRR7 gene deletion in vivo on excitatory synapses.
Based on the in vitro data, we developed a visual platform that could quantitatively depict in vivo degradation behavior of new injectable biomaterials by real-time and non-invasive fluorescence tracking.
Based on recent in vitro data, we tested the hypothesis that microarray expression profiles can be used to diagnose sepsis, distinguishing in vivo between sterile and infectious causes of systemic inflammation.
In view of the above in vitro data, we further tested the effects of this combination strategy in vivo.
Based on our in vitro data, we tested whether IL-2 or IL-12 were required for Tc17 plasticity in vivo, and found that neither were absolutely required.
Based on these in vitro data, we speculated that RNase 7 is secreted in vivo on the body surface.
Based on our in vitro data, we hypothesized that MGAS5005Δsrv would be largely unable to form biofilms in vivo.
Based on our in vitro data, we hypothesized that androgens might modulate ARG2 protein expression in PCa patients as well.
Based on these in vitro data we checked the in vivo effects of pantoprazole in a murine model of ovalbumin-induced allergy.
Consistent with our in vitro data, we also observed a decrease in T cell priming with co-administration of CD8α+ DC and monocyte (Figure 4E).
To evaluate possible clinical relevance of our in vitro data we also examined T-Ag expression in 33 sporadic CRC tumors and the associated liver metastases.
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