Sentence examples for vitro and zebrafish from inspiring English sources
Calycosin was shown to induce angiogenesis in human umbilical vein endothelial cell cultures (HUVEC) in vitro and zebrafish embryos in vivo via the up-regulation of vascular endothelial growth factor (VEGF), VEGFR1 and VEGFR2 mRNA expression.
Both in vitro and zebrafish xenograft model demonstrated the anti-lung cancer effect of BPIQ.
Both in vitro and zebrafish xenograft model were performed to examine the anti-lung cancer effect of BPIQ.
With human umbilical vein endothelial cells (HUVECs) in vitro and zebrafish in vivo models, PMFs showed different degrees of potency of antiangiogenesis activity.
Human FGF22 specifically bound to human FGFR2b in vitro and zebrafish fgfr2 are expressed in the midbrain during somitogenesis (Zhang et al., 2006; Ota et al., 2010).
The approach takes advantage of cell culture, in vitro, and zebrafish technologies, and is elegant, well-designed, and with appropriate controls.' The reviewers also raised the following major concerns that would need to be adequately addressed for the manuscript to be accepted: 1) The evidence that SerRS binds to the VEGF promoter and antagonizes cMyc appears to be convincing.
A ligand-free Pd-catalyzed C N cross-coupling/cyclization strategy has been developed for the rapid and milder access to this class of compounds some of which showed interesting pharmacological properties when tested in vitro and in zebrafish embryos.
The present study demonstrates that calycosin enhances endothelial cells proliferation in HUVECs in vitro, and in zebrafish embryos in vivo.
Although calpain inhibition promotes autophagy in-vitro and in zebrafish, it still remains to be determined whether it can be effective against HD in in-vivo mammalian models or in clinical settings.
This present study examines whether calycosin acts on ER and promotes angiogenesis in HUVEC cultures in vitro and a transgenic zebrafish model in vivo.
Accordingly, NRP1 knockdown and pharmacological CDC42 inhibition similarly impaired filopodia formation in vitro and in developing zebrafish in vivo.
