This longitudinal study and the two cross-sectional studies discussed above find a positive association between disease progression and virus variation [17], [18].
The great advantages of an immune response analysis during this time of the infection compared to the chronic phase, is that a lesser grade of virus variation might have occurred.
The bigger the proportion, the more the corresponding region accounts for virus variation and evolution.
It is likely that numerous factors are involved; the first is virus variation.
It is not known how much virus variation is regained when infecting U87.CD4 cells with primary isolates.
We investigated antibody prevalence, active infection, and virus variation in serum and cecal content samples from 629 pigs at slaughter.
He surely would have been in awe, as we all are, of the molecular explanation of influenza virus variation with succeeding epidemics.
Generally, a higher trunk proportion implies that the corresponding region accounts for more virus variation and evolution and that that region is more likely to be the source of the virus.
The investigation on molecular evolution of DEN-3 virus during the 1998 epidemic in Taiwan, currently in progress, will elucidate the possible role of virus variation in the pathogenesis of DHF.
Further analysis of the trunk proportion reveals a dynamic change of regions over time, suggesting that the contribution of each geographic region to virus variation and evolution changes annually.
