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Viral 5' UTRs are non-coding regulatory regions with potential roles in almost every step of viruses' life cycle and certainly relevant for viral fitness.
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Thus the virus life cycle should be cut off once the action of SAH is inhibited.
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Results with VSV/NL4-3 infection, suggest inhibition of both entry and post-entry events of the virus life cycle.
We investigated binding specificity of the mosaic virus and the possible interference of the two fibers during virus life cycle.
Viral replication by acquisition of the host cell biology represents a central part of a virus life cycle.
Viral protease 2A (2Apro) initiates the virus life cycle, and is an excellent target for developing antiviral drugs.
We have also excluded the possibility that both inhibitors act at the replication step of virus life cycle.
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