Sentence examples for viral proliferation from inspiring English sources

Vaccination has been the most successful weapon against viral infection; some infections may be treated with antiviral drugs or interferon (proteins that interfere with viral proliferation).

Thus the control of translation and replication during the initial period post-infection is essential for viral proliferation.

It is evidenced by severe inflammation to control initial burst of viral proliferation (represented in Fig. 9a).

We hypothesized that the Auger electron emitting isotope 125I can be used to control viral proliferation.

The association of RIG-I with pgRNA also leads to the induction of type III IFN, which plays a negative role in viral proliferation (Sato et al., 2015).

In many cancers, by comparison with normal tissues, the competency of the cellular anti-viral mechanism is impaired, thus creating an exploitable difference between the tumour and normal cells, as an unimpeded viral proliferation in cancer cells is eventually cytocidal.

For example, recent research has demonstrated that cancer utilizes exosome mediated transport in a unique manner to assist in tumor proliferation, viral exchange, microRNA transport, or exchange of signaling molecules or their breakdown products for communication of information among cancer cells and to aid the cancer community [120].

Whereas viruses also encode miRNAs that target specific host genes and pathways to prolong the longevity of infected cells or evade the immune clearance, which may be beneficial for viral infectivity and/or proliferation (Carl et al., 2013; Kincaid and Sullivan, 2012).

In a control experiment, the purified T cells were cultured in supernatant obtained from virus infected NSPCs, to rule out the effect of the viral antigen on proliferation of T cells (data not shown).

However, the intermingling of these clades with clades comprised of both genital and blood sequences and the absence of tissue-specific genetic features suggests compartmentalization between blood and genital tract may be due to viral replication and proliferation of infected cells, and questions whether HIV-1 in the female genital tract is distinct from blood.

Close inspection of the five subjects with statistical evidence of compartmentalization by three or more tests revealed pairs and small clades of genetically similar and/or monotypic (identical) viruses from the genital tract and/or blood (Subjects 4, 5, 8, 9 and 13 in Figure 1 4), suggestive of a burst of viral replication or proliferation of infected cells.