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Similarly, PiT1neo/Δ5 compound heterozygotes resulting from PiT1neo/+ and PiT1Δ5/+ matings were not viable (Table S3).
No homozygous Lrp1btm2wtsi mice were detected, suggesting that homozygous Lrp1b mice were not viable (Table 1).
Flies expressing all other mutant proteins or the wild type control were viable (Table 1).
Approximately equal numbers of wildtype and heterozygous Sdhd mice were obtained, indicating that these mice are fully viable (Table 1).
Despite the presence of Drosophila Lamin C aggregates, flies expressing rod domain amino acid substitutions were viable (Table 1) and showed no visible adult phenotypes.
Male flies inheriting roX1 MS2-6 roX2Δ chromosomes derived from three independent gene conversions were fully viable (Table 3).
Similar(49)
The result showed that after exposure of conidia to the inhibitor for 24 and 48 h, essentially all of them became non-viable (Table 3), indicating that the antifungal effect of the inhibitor was fungicidal.
In contrast, the O-TTG1 lines (with over-expression of BnTTG1 within the hairy leaf AtGL3+ B. napus recipient background) showed substantial changes to phenotype and were usually non-viable (Table 1).
All PCR positive and inconclusive samples were titrated, however none yielded viable virus (Table 1).
All crosses except one were successful and resulted in viable progeny (Table S15).
Protease treatment did not impair cell integrity, in terms of viable cells (Table 2).
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