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Since the major glutamate transporters EAAT1 and EAAT2 are also localized to astrocytic end feet [ 27, 28], it is a viable hypothesis that GAT-3 and EAATs are coexpressed on astrocytic end feet surrounding glutamat ergic synapses.
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It is, after all, only one of several viable hypotheses that are hotly debated among evolutionary biologists.
Reasons for the poor agreement are discussed; it is concluded that the only viable hypothesis is that mode shape statistics are not described by real Gaussian random functions.
One very viable hypothesis suggests that synaptic failure in AD is due to altered synaptic protein composition and function.
The most viable hypothesis is that the sensory nerves of the paranasal sinuses and nasal mucosa which are the branches of the trigeminal nerve (TN) may play a major role in AH.
One viable hypothesis is that, like Kif17, and perhaps Osm3, Kin5 transports specific membrane proteins such as guanylyl cyclase [1] into the Tetrahymena cilium.
This is truly a hypothesis that is extremely subjective.
There is an alternative hypothesis that makes far more sense.
Depending upon the situation, that B causes A might become a viable alternative to the hypothesis that A causes B but there is no reason to think that this alternative must always be the superior one.
Also, the hypothesis that viable count reduction would be greater at the anode was tested.
Evidence relating to a hypothesis that viable cells interact with one another as they do with lethally irradiated cells was conflicting.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com