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In order to verify of whether an indirect (HPMC-dependent) effects of stilbenes on angiogenic endothelial cell behaviour correspond to their direct activity, proliferation and migration of HUVECs, HMVECs and HMEC-1 cells were assessed in response to 48-h exposition to RVT, 3,3′,4,4′-THS and 3,3′,4,4′,5,5′-HHS.
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The primary aim of the study, however, was to verify whether administration of >75% of the prescribed CRRT dose to participants in each treatment arm was feasible.
Based on these findings, it should be of interest to verify whether patients of the low ratio group are more likely to be supervised by specific profiles of GPs.
We therefore created a phosphorylation-defective (S288A) and a phosphorylation-mimicking mutant (S288D) of that residue to verify whether phosphorylation of Ser288 might regulate UIM function.
Thus, we were able to verify reliability irrespective of whether we used the sequences of our ESTs or full-length cDNAs in the analysis.
This study aimed to verify whether administration of LMF stimulates expression of UCP-1, -2 and -3 in normal mice.
XPS measurement was performed to verify whether parts of the oxygen-containing groups of rGO underwent hydrophilic treatment.
To verify whether cleavage of compound occurs in the presence of live cells, we measured FRET changes in 5 in the presence of platelets by fluorescence scans.
This was used to verify whether diversity of health care philosophy and involvement of each team member increase when moving along the continuum (hypothesis Philosophy and values 1).
We also sought to verify whether overexpression of miR-22 would affect protein levels of the predicted target sTNFR1A (soluble tumor necrosis factor receptor 1A).
Next, we verify whether depletion of p21 could affect the inhibitory function of dsP21-397 in T24 and EJ cells.
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