Sentence examples for ventricular architecture from inspiring English sources

These fundamental architectural changes are then used to evolve new procedures that restore a more normal, helical, ventricular architecture in ischemic and dilated cardiomyopathy.

Aim of the study was to investigate exercise performance, left ventricular architecture and function, vascular reactivity, and carotid intima-media thickness in a group of patients with KS.

The activation of adrenergic system, endogenous vasodilators, the activation of the rennin-angiotensin system and changes in ventricular architecture whose major component is ventricular remodeling are among the most known regulatory mechanisms.

We evaluated cardiac function with echocardiography at 2 and 4 weeks after therapy, and analyzed cardiac angiogenesis and left ventricular architecture from histological sections at 4 weeks.

Ventricular architecture and the differentiation status of the myocardium are essentially maintained and are features of physiological hypertrophy [ 1– 3].

Remarkably, restoration of ventricular TG and ATP deficits via AMPK agonism as well as inhibition of gluconeogenesis improves ventricular architecture and function.

Topics discussed include molecular determination of cardiac phenotype (contractile and conducting), remodeling of ventricular wall architecture and its blood supply, and relation of trabecular compaction to noncompaction cardiomyopathy.

Our findings demonstrated that through incorporation of PGS, it is possible to create nanofibrous scaffolds with well-defined anisotropy that mimic the left ventricular myocardium architecture.

This study sought to determine the effects of abnormal left ventricular (LV) architecture on cardiac remodeling and clinical outcomes in mild heart failure (HF).

The objectives of this work are to evaluate a novel non Doppler-based echocardiographic method that makes it possible to simultanon Doppler-basedechocardiographicitudinal comethodthatf makesrditl velocity (V) and strain (S), and to assess whether left ventricular fiber architecture affects the net function of the myocardium.

The forebrain specific loss of early AspM expressing cells caused the elimination of most of the proliferating cells of brain, a severe derangement of the ventricular zone architecture, and the impairment of the cortical lamination.