However, few studies have been conducted to analyze the effect of first-order vein number on the transport efficiency of leaf vein networks.
The wide anatomical variability, e.g., in vein number, size, and anatomic connections, is probably largely responsible for the inconsistent occurrence of postoperative complications.
Surprisingly, several studies have failed to support the hypothesis that assessment of pulmonary vein number, location, and branching prior to RFA affects long-term efficacy of RFA [ 44– 44].
To address this question, we introduced descriptors of vein network topology that enable quantification of vein number, connectedness and continuity, and combined these topological descriptors with cellular imaging and molecular genetic analysis to quantify the contribution of PIN1, PIN5, PIN6, and PIN8 to vein network topology.
and P. tenuispadix E.G.Gonç., but it differs by its longer internodes, smaller leaf blade, number of primary lateral veins, number of inflorescences per sympodium, absence of extrafloral nectaries and spathe features (Table 1).
Additionally, we also evaluated the radiological findings such as the presence of extracranial metastasis and main portal vein thrombosis, number and type of HCC nodule, size of the largest HCC nodule, number of brain lesions, concomitant intratumoral hemorrhagic changes and location of the brain lesion.
In a similar vein, the number of CD3+, CD8+, and CD25+ lymphocytes (as well as mast cells) has previously been found to be elevated in the colonic mucosa of a subgroup of those with IBS [49].
NOTCH1 is considered an important therapeutic target for CSCs, and in this vein, a number of NOTCH1 inhibitors are being developed (Al-Hussaini et al, 2011).
Average access time (skin to vein) and number of attempts were significantly reduced in the ultrasound group of patients compared with the landmark group (p < 0.001).
Preprocedural identification of pulmonary vein anomalies their number, location, or branching could potentially decrease the recurrence rate of AF and the risk of pulmonary vein stenosis.
In a similar vein, a number of studies have suggested that MMR deficient cells, including MLH1 and PMS2, produce more methotrexate resistant colonies than MMR proficient cells (Frouin et al, 2001; Lin et al, 2001; Snijders et al, 2008).
