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Such variations are thought to be attributable to the Milankovitch cycle, which describes changes in the path of the Earth around the Sun.
Four of those variations are thought to cause gum disease.
These variations are thought to reflect natural flexibility at this site required for insertion into the peptidoglycan mesh.
In humans, these slow BOLD variations are thought to reflect an underlying or intrinsic form of brain functional connectivity in discrete neuroanatomical systems.
However, leptospiral LPS is highly variable; its variations are thought to be the major antigenic determinant defining differences between approximately 230 serovars and contributing to serovar specific immunity [7].
Recurrent somatic variations are thought to be drivers but they are present in only a small fraction of patients.
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Variation in gene transcript levels is thought to be an important modulator of disease risk in humans and SNPs that may mediate this variation are thought to be of great functional significance [ 26].
These two types of variation are thought to be implicated in malignant transformations for many (if not all) types of solid tumors.
Genes that display limited intra- and inter-species variation are thought to have undergone positive selection pressure directed towards a functional conservation and may have played a pivotal role in species evolution.
The knockout variation is thought to be due to stochastic epigenetic variation present in the early embryo.
This variation is thought to play an important role in the muscles' ability to produce high power, at least in elite athletes [27].
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