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The causative genetic variation was mapped by linkage analysis in a chromosome region containing the Mc1r locus.
The locus PH24, explained 10% of phenotypic variation, was mapped to a small region on Gm19 where LD decayed rapidly.
The geographic distribution of the mtDNA variation was mapped and the genealogic relationships of haplotypes were inferred using the median-joining algorithm implemented in Network 4.5 [ 92].
The causative genetic variation was mapped by linkage analysis in a chromosome region containing the Mc1r locus, but subsequent mapping of an Mc1r marker showed that the gene is most likely not responsible for the color difference in N. furzeri [ 20, 21].
A major QTL, explaining 88% of the total phenotypic variation, was mapped to a chromosome region that spanned 178 cM and contained 205 GBS markers plus 1 SSR marker and 1 gene marker, with 0.86 cM per marker in genetic distance.
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The geometry variation and spatial strain hardening variation were mapped onto the model.
The contour maps illustrate the regions where the sheet resistances of each bubbler pressure variation are mapped.
Sharp gray level variations are mapped to positive (white) detail image values.
The detail signal is almost zero at areas of smooth gray level variation and sharp gray level variations are mapped to positive detail signal values (white).
All variations were mapped to all transcript models, which led to multiple annotations for several loci.
The variations were mapped to the respective genes or intergenic regions using the published annotation of the reference genomes Sterne (NC 005945) for 34F2 and its derivatives and Ames Ancestor (NC 007530) for ΔANR and its derivatives by Annotation Miner software (https://sourceforge.net/projects/annotationminer/).net/projects/annotationminer/
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