In all mice tested the editing level of GluR2 Q/R site was virtually 100%, with no variations due to either KA injection or genotype (data not shown).
Much of this variation is attributable to either measurement error or variation in TL between cells in the same individual.
Allelic variation may lead to either gain or loss of function through the IL-4R.
If however, such variation arises due to either stochastic errors or environmental influences acting after development of the villus tree structure, there may not be a significant intra-site correlation.
Nucleotide changes known as mutations or variations can lead to either gain-of-function or loss-of-function of oncogenes and tumor suppressor genes.
This variation can be attributed to either intrinsically random processes, genetic variation, or environmental differences within the population.
Variation can be due to either regulation by transcription factors or by epigenetic factors.
Theoretically, genomic variations would give rise to either ~ 50% or ~ 100% mismatch ratios depending on whether the variation is heterozygous (Additional file 6A) or homozygous (Additional file 6B) [ 33].
A flow asymmetry can appear due to either variations in the solar wind dynamic pressure or inherent features of solar wind/Mars interaction.
