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On average, 241 missense somatic variants were analyzed per sample.
All collagen variants were analyzed for their ability to assemble into D-periodic fibrils.
The clones and blood bulk DNA from the donor were sequenced, and the somatic variants were analyzed as a control for the in vitro-induced mutations.
The relationship between the structures and the activities of the variants were analyzed by modeling the structures of the endoglucanase II variants.
Finally, the enzymatic properties of recombinant variants were analyzed, and compared to those of reAoXyn11A.
On the economical level, four variants were analyzed: earth dam with two types of spillways (lateral and frontal), fully submersible RCC dam and incorporated spillway on the dam.
MAIN OUTCOME MEASURE: Germline variants at any site in the coding sequence, splice junctions, 5' untranslated region, or 3' untranslated region of the BRCA1 gene were analyzed in cases, and selected variants were analyzed in controls.
The putative pathogenic mechanisms of the novel variants were analyzed in silico.
19 variants were analyzed in relation to 31 phenotypes that were assessed in a health interview and examination study.
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In total, 1010 deleterious natural splice site variants were analyzed (Supplementary Table 4).
All available sequences of viral variants were analyzed.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com