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Human studies suggest that these NOD2 variants result in a loss of function [74].
The fluctuation of markets and increasing product variants result in uncertain production volumes.
Hilar biliary anatomical variants result in a range of unusual but predictable patterns of segmental atrophy with or without hypertrophy.
Transcript variants result from alternative promoters usage and encode for the same KLK6 protein, since all mRNA sequence changes occur at the 5′-untranslated region, while splice variants result mainly by skipping coding exons and they either encode for small proteins with low identity to KLK6 or to no proteins at all [6, 7].
Possibly, IL23R variants result in alternative mRNA splicing, leading to corresponding IL-23R isoforms [46] with different tissue distributions.
All three variants result in significantly reduced RPS19 levels when compared to the corresponding wild type sequence.
Similar(19)
Column "×" shows that the number of inactive variants resulted from amino acid substitutions.
Progressive accumulation of antigen loss variants resulted in a viral preparation with lower immunogenicity after serial passage.
C-terminal truncation of the full-length peptide variants resulted in reduction in salt-tolerability of the microbicidal effect as well as in reduced anti-inflammatory properties.
The two remaining coding variants resulted in synonymous changes.
Selection for such variants results in higher mutation and recombination rates.
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