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Its risk of being overwhelmed by variants, is, in principle at least, related to this total of 10^13.
One of the new variants is in a gene active at synapses, the junctions between brain cells, and the two others help damp down inflammation in the brain.
One of these variants is in the empiricist tradition.
The significant presence of interconnected cytoskeletal, signaling, and secreted proteins among the variants is in agreement with the notion of increased platelet activation in the NSTE-ACS patients in parallel to the acute event.
However, our understanding of the biological mechanisms behind these genetic variants is, in many cases, incomplete.
The question of determining the potential pathogenicity of mtDNA variants is in fact a matter of debate among clinicians (McCormick et al. 2013).
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Estimates from the method are valid even if the genetic variants are in linkage disequilibrium.
Successor variants are in the inventories of both Russian and Ukrainian air-defense units.
The variants were in the PCSK9 gene and in HMGCR, which encodes the target of commonly used statin drugs.
Our measurements of these variants were in agreement with the expectations based on DMD (Fig. 5 and Supplementary 11).
Indeed, many of the germ-line variants were in genes related to DNA repair, microsatellite instability, and chromosome instability.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com