Exact(6)
Comparison of the frequency of variants differed from EP in both groups of patients (P=0.01) with an odds ratio (OR) of 5.14 (CI 95% [1.07 26.56]).
FMS patients and family members without rare variants differed from control subjects with regard to both TH1 (IFNγ) and TH2 (IL-5 and IL-13) cytokine levels (Figure S2).
Each of the variants differed from the original MCF-7 line in ploidy, modal cell volume, and signaling pathway usage.
Moreover, each of these rare variants differed from all other sequences by a single non-synonymous (amino acid altering) substitution at an otherwise invariant site.
The in vitro and in vivo phenotypes of cells expressing glycosylation site variants differed from cells expressing fully-glycosylated ICAM-2 or no ICAM-2.
The 2 variants differed from each other by 0.5% of nucleotides, and their neuroinvasiveness in mice was similar to that of the New York 1999 isolate.
Similar(54)
Assembly of the 5S rRNA gene confirmed three variants (data not shown); downstream of the 5S rRNA gene, all three variants differ from Methanoculleus marisnigri JR1, indicating a different genomic context for the rrn cluster.
These variants differ from one another by the presence of either C or T nucleotide at codons 112 and 158.
CenH3 variants differ from the other H3 variants by a long extension of the N-terminal tail, which are not conserved among eukaryotes [ 5, 6].
Variants differ from canonical histones in their protein-protein interactions, localization in chromatin, number and types of PTMs, nucleosome stability, and tissue-specific expression profiles [ 5].
Most of these variants differ from each other at the second (X) and/or fourth (Z) amino acid position in the cyclic heptapeptide.
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