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The former variant is shown to require the least accommodation strain within the parent grain and to have the greatest potential for growth.
The ability to cleave the receptor by Urokinase-type Plasminogen Activator (uPA) and the generation of the A6p structural variant is shown schematically (Fig. 1c).
The design of each truncated variant is shown in the cartoon below.
The fluorescence intensity on a single bead over time for each p62 variant is shown.
In contrast, if a single new missense variant is shown to have arisen de novo, then it is possible that it may be a pathogenic autosomal dominant mutation.
To our knowledge, this is the first time that an U1 snRNA variant is shown to correct exon skipping due to polypyrimidine tract and exonic mutants.
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By using recombinant DNA techniques, three of the splice variants were synthesized in Escherichia coli and each variant was shown to stimulate both adenylyl cyclase and calcium channels.
The potential use of this variant was shown by reducing the size of the weld root defect, even for significant levels of lack of penetration, without affecting overall metallurgical characteristics of the welded joints.
Representative immunoblots of each variant are shown.
This variant was shown to be responsive to TSA treatment in our experiments.
Subsequently, the R141E/Y288A/A306F variant was shown by analytical ultracentrifugation to be partially dimeric.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com