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The validity of this mechanism has attracted national attention.
Here we evaluate the validity of this mechanism and the influence of alkyl-chain length by extending our work to a series of primary alcohol molecules: methanol (CH3OH), ethanol (CH3CH2OH), and 1-propanol (CH3CH2CH2OH).
However, past studies focused on the peak velocity of SAPS for data selection and did not confirm the validity of this mechanism.
To confirm the validity of this mechanism, we first attempted cloning of a simple test gene, lacZα, into a modified pUC19 expression vector (see Methods S1 for sequence information).
In primary HBE cells, EGTA eliminated MMP-1 secretion in response to DEP or OE, thus confirming the validity of this mechanism.
Further investigation of the effects of the nonsynonymous SNPs on Tcf19 function will likely yield more insight into the validity of this mechanism of disease susceptibility.
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Future research could usefully examine the validity of this possible mechanism in an ecologically valid setting.
To assess the validity of this reaction mechanism in non-premixed systems, extinction strain rates of DME air mixtures, which are not available in the literature, are measured in a counter-flow diffusion flame burner as a part of the present work.
In this section, we provide further evidence on the general validity of the mechanism discussed above for other European countries besides Spain.
Having tested the validity of the mechanism of Fig. 1A as a quantitative description of actin cable dynamics, we now use the model to describe the response of the system to changes in parameter values in order to (i) suggest experiments for further tests of the model, and (ii) provide insights on the biological mechanisms of actin cable control.
The results presented here demonstrate the increment of labile zinc levels in the presence of cadmium, supporting the validity of the mechanism proposed above.
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CEO of Professional Science Editing for Scientists @ prosciediting.com