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Utilizing this model we have derived several conclusions: i) Both redox and non-redox inhibitors of 5-LO decrease LTA4 production independently of lipid peroxide level.
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Utilizing this model for structure-function analysis, we identified and characterized a gain-of-function mutant of FIT2 (FLL(157-9)AAA) 157-9 AAA 157-9 AAAmain 4 transmembrane augmentedomaintotal number and mean size of lipid droplets.
We utilize this model for the purpose of cluster validity assessment as well.
We utilized this model within an inverse problem framework to estimate the hyperelastic parameters of adipose tissue extracellular matrix (ECM).
We utilize this model to determine the time course of articular cartilage and subchondral bone changes following knee injury.
We utilized this model to quantitatively assess the relationship between adhesive forces derived from cell substrate interactions and the ability of fibronectin fibril assembly to induce cellular self-assembly.
We utilized this model to directly test the functional role of HIF-1α in transcriptional modulation during RSV infection.
Here we utilized this model of PLN-induced lung inflammation to determine the contribution of TLR2 and TLR4 to PLN effects in vivo.
Therefore, we utilized this model of neural differentiation to study the mechanisms underlying the differentiation of hPSCs towards NPCs.
We utilized this model to demonstrate that HLF decrease intravascular CH levels and interact with cholesterol to improve CO in diseased fish.
We utilized this model for the study of infection by M. marinum showing that injection of this pathogen in the tail fin results in a local infection, which leads to the formation of single granuloma-like structure.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com