In comparison to other network transfers, for the first time we utilized two model organisms resulting into a more diverse and complete network of the target organisms.
The first experiment utilized three rigid models of the tibiofemoral joint to assess the noise and to compare digitization errors of two independent examiners.
For this experiment, we utilized three different models of frataxin deficiency.
Herein, we have utilized two cellular models of EOC, where transfer of normal chromosome 18 material into the EOC cell lines TOV-112D and TOV-21G induced in vitro and in vivo suppression of their tumourigenic phenotype.
To this end, we utilized two independent models of experimental hepatitis induced either by concanavalin A [ConA, that triggers autoimmune hepatitis (Tiegs et al, 1992)] or carbon tetrachloride [CCl4, that causes hepatic necrosis and reactive hepatitis (Luster et al, 2001)].
These studies utilizing three murine models of different types of B-cell lymphoma collectively demonstrate in vivo activity of AR-42.
The model utilized three sets of groups; mono-cultures of either M. viscosa or A. wodanis in one tube was implanted, the second set where M. viscosa and A. wodanis were implanted together in separate tubes, and the third set where M. viscosa and A. wodanis were implanted together in the same tube.
In this study we utilized two murine models to compare the growth of large peritumoral lymphatic and blood vessels.
We utilized two infection models that differ in the intensity, onset and duration of antigen-presentation and inflammation.
Collectively, robust phylogeny estimation, which utilized three separate optimality criteria and various models of protein evolution, consistently placed T. equi as a sister taxon to the Theileria clade, with all three of these piroplasm species subtended by B. bovis.
