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We utilized a tissue microarray of 240 primary untreated and unselected breast cancers.
In order to relate our in vitro findings to actual human patient tumor specimens, we utilized a tissue microarray (TMA) to evaluate STAT6 expression in GBM, healthy brain, and lower grade gliomas by immunohistochemistry (IHC).
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Next, utilizing a tissue microarray we simultaneously evaluated 107 surgically resected human pancreas cancer specimens for CEA expression with IHC to validate previous reports and compare with our xenograft models.
In the current study, we expanded these results utilizing a tissue microarray.
Overexpression of Mcl-1 was found in malignant melanoma compared to benign nevi and increased expression of Mcl-1 was also observed by comparing primary and metastatic melanoma samples utilizing a tissue microarray [ 6].
We utilized a tissue-microarray (TMA) containing specimens from benign prostatic tissue and malignant tumors from 448 patients who underwent radical prostatectomy for localized prostate cancer.
We conducted the search for polySia-NCAM and NCAM by utilizing a tumor tissue microarray (TMA) of the 36 paraffin-embedded neuroblastoma samples, as described earlier [ 24].
A tissue microarray (TMA) from the Samsung Medical Center Breast Cancer Biomarker Study was utilized for the analysis of FOXM1 status.
Expression of CEA on human pancreas cancer specimens was evaluated by performing IHC utilizing the anti-CEA scFv-Fc (H310A) antibody fragment upon a tissue microarray containing 107 1-mm-tissue cores of pancreatic adenocarcinomas.
A tissue microarray of these 1420 patients was constructed.
A tissue microarray block containing 66 HCC tissues was constructed by using a tissue microarrayer.
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