Sentence examples for using truncated forms of from inspiring English sources

Exact(7)

Contrast media could be designed using truncated forms of these proteins.

For application to heterogeneous porous beds, the assumption of weak spatial variation of the porous resistance is examined using truncated forms of the governing equations.

Similarly, by using truncated forms of CLIP-170, we found that both the coiled coil domain and the zinc finger domain of CLIP-170 were necessary for its interaction with HDAC6 (Fig. 5E).

In this study we designed recombinant vaccine against urinary tract infections (UTIs) using truncated forms of FliC and type 1 fimbrial FimH adhesin from uropathogenic Escherichia coli (UPEC) and studied their in silico interactions with Toll-like receptor 5 (TLR-5) via docking protocols.

Our finding shows that soluble gp91phox 221-C displans an NADPH- and a p67phox-dependent diaphorase activity (Figure 3B and 4B) identical to that described by Nisimoto using truncated forms of gp91phox over-expressed as inclusion bodies in E.coli [6].

Using truncated forms of proteins, the affinity for Ca2+ was measured to be 60 μM to 3.8 mM, a range that exceeds the affinities for Ca2+ activation.

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Similar(53)

We used truncated forms of μ1 in our experiments, because of their better expression and solubility than the full length μ1 (residues 1 423).

To evaluate whether the RS2 segment functions as a molecular trigger for activation through the NRF or possibly through other sequences in the SID, we investigated SR RS2 -dependent changeSR RS2 -dependentylation of SR(ΔRS2) using several trunchangesorms of SRPK1.

To establish whether sequences inside or outside the kinase domain are important for RS2-dependent activation, we investigated how SR RS2) affects the phosphorylation of SR ΔRS2) using several truncated forms of SRPK1.

Using various truncated forms of the U4/U6 snRNA duplex for unwinding assays Mozaffari-Jovin et al. [ 52] concluded that Brr2 binds to the 3′ end and translocates along the U4 snRNA.

Using various truncated forms of FHL1C, we found that the RBP-J-binding motif of FHL1C had almost the same effect as full-length FHL1C on the induction of apoptosis, suggesting that the minimal functional sequence in the RBP-J-binding motif of FHL1C might be a new drug candidate for T-ALL treatment.

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