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Thus, to obtain functional insight into Spp1 in the end-stage liver disease HCC, we analyzed the mRNA level using the human cancer microarray database Oncomine.
Using the human cancer cell xenograft model, we were able to quantify lung metastases 4 weeks after the surgical resection of primary tumors.
Using the human cancer genome atlas database and two independent breast cancer cohorts we find that L1CAM is inversely correlated with androgen receptor (AR) expression.
In our experiments using the human cancer cell (HeLa S3) cycle data (Whitfield et al., 2002), available at http://genome-www.stanford.edu/Human-CellCycle/Hela/, we apply our method to three different datasets, corresponding to three experiments under basically the same conditions but having different sample sizes (each with 12, 26 and 48 time points).
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Hence, we first determined in vitro cellular uptake and transgene expression using the human lung cancer cell line, A549, as a cancer model.
We evaluated the effect of anti-miR-142-3p anti-miR-142-3p anti-miR-142-3p anti-miR-142-3pnd confirmed that the protein level of APC was elevated in the husingbreastheancer xenograft cells transfected withuman anti-miR-142-3p-expressing lentivirus.
However, one report using the human breast cancer line MCF7 has shown that the SP phenotype can be used to identify cells with characteristics of cancer stem cells that express the tumour antigen MUC1, supporting a role for the SP in further analysis of human BCSCs [ 115].
The regulation of PRAP1 expression by cellular stresses was studied using the human colon cancer cell line, HCT116.
We tested this hypothesis using the human breast cancer cell lines MCF-7 and MDA-MB-231.
PTEN restoration was tested in a xenograft model using the human prostate cancer cell line, PC-3-Bcl-2.
The EGFR-dependent human tumor xenograft model was established in mice using the human epidermoid cancer cell line A431 (European Collection of Cell Cultures).
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