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All imaging and biodistribution studies were performed using the human breast carcinoma, ER-positive cell model ZR-75-1 purchased from ATCC (Manassas, VA).
We tested this hypothesis using the human breast cancer cell lines MCF-7 and MDA-MB-231.
In an experimental study, the subcellular localization and function of these molecules was examined by using the human breast cancer cell lines MCF7 and SK-BR-3, both of which highly express S100A14 and S100A16 proteins.
Therefore the present study aimed to investigate the effects of chronic glucose deprivation on ALA-induced photodynamic therapy, the rate of intracellular PpIX generation, extra-cellular PpIX accumulation and PDT sensitivity, using the human breast cancer cell line MCF-7.
Although we could not perform TOP/FOPflash experiments in the MMTV-Wnt1 tumors using the lentivirus with the TOP/FOPflash constructs, we evaluated the effect of anti-miR-142-3p anti-miR-142-3p anti-miR-142-3p anti-miR-142-3p
We determined estrogenic activities of 17 prevalent PCB congeners using the ER-CALUX bioassay (BioDetection Systems) using the human breast carcinoma T47D.Luc cell line, stably transfected with pEREtataLuc construct (Legler et al. 1999; Machala et al. 2004).
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We have here used the human breast adenocarcinoma MCF7 cell line to study the role of flotillins in breast cancer signaling.
We used the human breast cancer cell line MDA-MB-231 to make orthotopic xenografts and grew them to an average tumour volume of 1.5 cm.
For this study we used the human breast cancer cell lines MCF-7, T-47D, MDA-MB-468 and MDA-MB-231 (ATCC, Manassas, VA, USA).
In order to establish if this resistance can be reversed in model systems, we have used the human breast adenocarcinoma cell line, MCF-7, that, like many of the breast tumours we have examined, expresses very high levels of MGMT.
The assay uses the human breast cancer cell line MCF-7 stably expressing GFP-LC3B to quantify autolysosome formation measuring GFP fluorescence intensity in 96-well plates, and identified 38 compounds as potential activators and 36 as inhibitors of autophagy.
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