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This research primarily was associated with fabrication of PLA-TPU nanocomposites using a low weight fraction of cellulose nanofibers as a biodegradable reinforcement.
The focus of the present work is to fill the space between the MNTs using a low weight percent of f-MWCNTs within the polymer matrix and thereby making utmost use of the advantages of f-MWCNTs and eventually achieve low-cost and improved EMI shielding materials.
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With a mass ratio of five and using a low molecular weight, a sequestration of 52.7 % was attained (run 9), which was higher than with high molecular weight chitosan (run 4).
Genexol®-PM was newly developed by using a low molecular weight, nontoxic and biodegradable amphiphilic diblock copolymer, monomethoxy poly ethylene glycol -block-poly d,l-lactide) (mPEglycol -block-poly dxel (glycol -block-poly dnex Co., Seoul, Korea).
Although this matrix is specific for positive ionization, data were acquired in both the positive and negative reflectron mode between 200 and 2000 m/z in an AutoFlex MALDI-TOF mass spectrometer (Bruker Daltonics), using a low molecular weight peptide mixture (Peptide Mix-5, LaserBioLabs, Sophia-Antipolis, France) for calibration.
The running buffer was 20 mM HEPES, pH 7.4, 600 mM NaCl, and the protein molecular mass was estimated using a low molecular weight calibration kit (GE Healthcare).
This work was designed to investigate the feasibility of using a low molecular-weight epichlorohydrin/dimethylamine (epi/DMA) polymer as a primary coagulant for the pretreatment of terrestrial humic-rich water prior to the microfiltration (MF) process, without removal of the flocs produced via the coagulation pretreatment.
Examination of molecular weights of extracted proteins was accomplished using a low range molecular weight marker (14 - 66 kDa).
The gel filtration column was calibrated by using a low-molecular-weight calibration kit (GE Healthcare) according to the manufacturer's guidelines, allowing determination of the molecular mass of eluted proteins.
Using a low-molecular-weight compound that competes with p53 for binding into a hydrophobic cleft on the surface of MDM2, termed nutlin-3 [ 122], we have been able to elicit a robust and selective activation of the p53 pathway in NB cells with wild-type p53, leading to G1 cell cycle arrest, apoptosis, premature senescence and neuronal differentiation [ 123].
The risk of bleeding may be minimized by using a low dose of high-molecular weight heparin (HMWH), the use of low-molecular weight heparin (LMWH) or regional anticoagulation with citrate to prevent clotting in the extracorporeal circuit (ECC) [ 7].
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