To explain the observed pH and spermidine concentration dependence, two different theoretical approaches were used: a chemical model of the charge equilibrium of spermidine and Monte-Carlo simulations of the orientation of the rodlike spermidine molecules in the gap between the borosilicate and mica surfaces.
The kinetic analysis was carried out to clarify the experimental phenomenon using a chemical kinetic model in Chemkin-Pro Softwith coupled with a PAH formation mechanism proposed by Appel et al.
In this way, the effect of fuel concentration (0.75% and 1.75%), pressure (2.0, 20, and 40 atm) and equivalence ratio (0.5, 1.0, 2.0) on ignition delay times was studied experimentally and computationally using a chemical kinetic model.
Therefore, the objectives of this study were to (1) determine sow posture frequencies and duration (2) ascertain the postural sequence and time when standing to lying and vice versa and (3) to record time spent for sows to access feed when lameness was induced using a chemical synovitis model.
This paper revisits the origin of dust in galaxies, in particular, in the Milky Way, by using a chemical evolution model of a galaxy composed of stars, interstellar medium, metals (elements heavier than helium), and dust.
This paper investigates the main driver of dust mass growth in the interstellar medium (ISM) by using a chemical evolution model of a galaxy with metals (elements heavier than helium) in the dust phase, in addition to the total amount of metals.
Using a chemical transport model [12] to measure PM2.5 concentration around the participants' residential areas, a 5% higher lung cancer mortality risk was observed for a 10 µg/m increase in PM2.5 concentration (95% CI: 1.01 1.10) [1.01 1.10
van Donkelaar et al. (2010) related AOD to near-surface PM2.5 by using a chemical transport model that predicts the state of the atmosphere from meteorologic data sets, emission inventories, and equations that represent the physical and chemical evolution of atmospheric constituents.
Recent studies by using a chemical carcinogen (diethylnitrosamine) model of HCC in mice showed that the gender disparity in HCC was owing to suppression of interleukin-6 production in Kupffer cells by oestrogen (Naugler et al, 2007).
