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A number of studies have used depression of activity (e.g., locomotor activity, feeding, rearing) to assess pain resulting from headache in humans [18] and rodents [7, 9, 17, 20, 28].
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We measured depression using the depression subscale of the Hospital Anxiety and Depression Scale (HADS-D).
To assess depressive mood we used the depression subscale of the Hospital Anxiety and Depression Scale (HADS-D) [ 7, 8].
We used the depression subscale of this comprehensive psychology scale.
Unlike previous studies we required a diagnosis of depression for study entry and we used remission of depression as a clinically relevant outcome criterion.
However, there is evidence supporting the use in depression of the EQ-5D-3 L (3-level where each question has 3 possible answers).
Accordingly, ranolazine produced a use-dependent depression of several Na channel parameters which can be also found in human atrial myocytes even at higher stimulation rates.
We adjusted for covariates previously used in EXPLORE analyses of HIV seroconversion, including race/ethnicity, numbers of sexual partners, self-report of sexually transmitted infections, methamphetamine and heavy alcohol use, depression, and use of drug and alcohol during sex.
Secondary outcomes include measures of substance use, depression and anxiety, quality of life, health care consumption and neuropsychological functions.
6/23 (26%%) studies used measures of depression, anxiety and/or stress.
This result disagrees with Boudreau who reported improvements of the same parameters we used, out of depression.
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