His work began during his graduate studies at Yale where he used a rat model for the experimental determination of brain glucose transport kinetics, energetics, and neurotransmitter metabolism.
This study used a rat model of adolescence to provide information regarding possible sex differences in contributors to social drinking.
This study used a rat model of post-traumatic knee osteoarthritis (OA) created by anterior cruciate ligament transection with partial medial meniscectomy (ACLT + pMMx).
The authors used a rat model of surgical acute ischemic nephropathy to test the hypothesis that fenoldopam attenuates ischemia/reperfusion (I/R)–induced NF-κB-mediated inflammation.
For this purpose, we used a rat model of TDP43-induced paralysis56,57,58,59. Human WT TDP43 cDNA was delivered to neonatal rats via an intravenous injection of recombinant adeno-associated virus serotype 9 (AAV9), yielding consistent limb paralysis that gradually progresses over the ensuing 2-16 weeks57,59.
To more fully explore the role of TGF-beta ligands, receptors, and potential modulators during skin development and wound healing, we have used a rat model that transitions from scarless fetal-type repair to adult-type repair with scar between days 16 and 18 of gestation.
We used a rat model to measure the secretory osteocalcin level in culture medium to verify that monitoring osteocalcin levels enables the selection of constructs with high osteogenic potential.
In this study, we used a rat model of chronic migraine to evaluate depression and anxiety behaviours in rats, and created scales for these affective disorders.
They used a rat model of Parkinson's disease in which a lesion is made to a group of dopamine-sensitive neurons on one side of the brain.
The authors used a rat model of dermal wound healing to assess the effects of fasudil with regard to the prevention of scarring.Human scar tissue and surrounding normal skin were immunostained for RhoA and Rho-associated kinase.
Coppey and co-authors used a rat model for T1DM with a dietary enrichment of menhaden oil, a natural source of n-3 fatty acids, that prevented and reversed multiple pathological endpoints associated with DPN [66].
