We modeled log BPA urine concentration as a function of fasting time, adjusted for urine creatinine and other confounders, in 1,469 adult participants in the 2003 2004 National Health and Nutrition Examination Survey.
The calculation is adapted from an equation that expresses the rate of urine flow as a function of AVP in humans [ 6].
This conclusion was based on statistics gathered from the Center for Disease Control and Prevention's (CDC) 2003 to 2004 National Health and Nutrition survey (NHANES), whereby the authors modeled BPA urine concentrations as a function of fasting time.
We evaluated variability in measures of dilution in spot urine samples across pregnancy by plotting urinary creatinine concentrations and urine specific gravity as a function of gestational age.
In addition, micro vascular changes were determined in the kidney through possible morphologic changes and by estimating urine protein/creatinin ratio as a function of renal barrier properties.
The chemical status of the radioactive compounds in blood and urine was analysed as a function of time using HPLC techniques.
Given this additional information, the authors explored whether systematic variations exist in UFR (measured in milliliters of urine produced per hour) as a function of age, sex, race/ethnicity, and body mass index (BMI) category.
Within the context of model assumptions, urine concentrating capability is increased by exponential decline in loop population as a function of medullary depth and by solute efflux localized near loop bends, in accordance with earlier, but less comprehensive, studies.
Final four-week abstinence was tested as a function of treatment arm, baseline abstinence (i.e., baseline urine drug and breath alcohol negative), CJS status, and time (Table 4).
Finally we examined the ORs for having a short AGD compared with a long AGD as a function of quartiles of phthalate metabolite concentration in the pregnant women's urine, with the lowest quartile as a reference.
