Sentence examples for uptake deficiency from inspiring English sources

Thereby, an astrocyte glutamate uptake deficiency triggers an increase of the (v_{1}) value, and we observe an increase in the oscillation frequencies in the neural time series.

Note that this value can be tuned to obtain (v_{1}^) small enough and (p_{{mathrm{SNIC}}}) large enough so that the frequency after regulation is equal or lower than the one before uptake deficiency.

Hence in this scenario, when (v_{1}) increases as a consequence of the astrocytic glutamate uptake deficiency, we expect to observe a decrease in the oscillation frequency in the neural activity.

Moreover, in Sect. 3 about the astrocyte GABA uptake deficiency, we proved that, for a fixed value of (v_{1}), (p_{{mathrm{SNIC}}}) is linear and increasing with (v_{2}).

Based on the interpretation of the aggregated yet biophysically significant parameters of our model we have reproduced in silico both glutamate and GABA uptake deficiency in astrocytes and illustrated by numerical simulations the different types of change in the neural activity resulting from such deficiency.

At any rate, it cannot be excluded that the impaired survival in CDM and virulence defect of the trkH mutant might not be totally attributable to its potassium uptake deficiency.

As mentioned in the introduction, these concentrations have been proven to modulate the local neuron excitability and this feedback has been identified in recent studies [1] to be an essential mechanism of several pathologies triggered by astrocytic uptake deficiencies.

Interestingly, when grown on the medium with low K+ level, only the E. coli transfected with OsGLR2.1 and OsGLR3.2 survived, indicating a complementation of OsGLR2.1 and OsGLR3.2 to the K+ uptake-deficiency of LB650 (Additional file 7B).

In addition, research reports suggest that continued application of nitrogen and phosphorus alone would accentuate the uptake and deficiency of other nutrients that are not supplied in the fertilizer.

DHA exhibited ROS-independent antitumor mechanism by down-regulating cell-surface TfR1 level through an unexpected endocytic pathway, leading to the decline of TfR1 mediated iron uptake and deficiency of cellular iron stores [120].

In addition, continued use of DAP and urea seems to have contributed to increased exchangeable acidity as well as accentuating the uptake and deficiency of other nutrients such as K and Zn that are not supplied in the fertilizer application.